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Muscle Inflammaging: The NLRP3 Inflammasome Contributes to Sarcopenia

dc.contributor.advisorSchertzer, Jonathan D
dc.contributor.authorMcBride, Marin Jane Finola
dc.contributor.departmentMedical Sciences (Cell Biology and Metabolism)en_US
dc.date.accessioned2016-09-23T18:50:22Z
dc.date.available2016-09-23T18:50:22Z
dc.date.issued2016-01-12
dc.description.abstractSarcopenia is the progressive loss of muscle mass and deterioration of muscle function during aging. This can affect quality of life and ability to perform daily tasks. During aging, muscles become weaker, slower, and have a reduced ability to generate energy quickly through glycolysis. It is unclear why this happens, but chronic inflammation during aging may contribute to these aspects of sarcopenia. NOD-like receptor protein 3 (NLRP3) increases caspase-1 activity, which is a part of the immune system involved in multiple age-associated diseases. We determined the role of NLRP3 in sarcopenia using adult and old mice.en_US
dc.description.degreeMaster of Science (MSc)en_US
dc.description.degreetypeThesisen_US
dc.identifier.urihttp://hdl.handle.net/11375/20448
dc.language.isoenen_US
dc.titleMuscle Inflammaging: The NLRP3 Inflammasome Contributes to Sarcopeniaen_US
dc.typeThesisen_US

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